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The study tested only leg exercises, but you can safely expect increased reps on other exercises, as well.

Citrulline? Yes thats the stuff the supplement industry claims to be a better version of arginine. A "super pump supplement", but not necessarily an ergogenic - in spite of the fact that corresponding evidence from rodent and human studies (Briand. 1992; Pérez-Guisado. 2010; Giannesini. 2011) existed years before the study at hand was published (Wax. 2014).

Benjamin Wax and his colleagues from the Mississippi State University and the Auburn University  investigated the effects of citrulline malate supplementation on lower-body resistance exercise performance, blood lactate, heart rate, and blood pressure.

Based on citrulline malate’s chemical composition and a review of the current literature Wax et al. hypothesized that citrulline malate supplementation would mitigate fatigue occurring to the working muscle; therefore, augmenting resistance training performance.
To test their hypothesis the researchers recruited 12 advanced resistance trained male subjects (85kg body weight; <12% body fat; 22.1 ± 1.4 years) and conducted a randomized, counterbalanced, double blind study.

The subjects were randomly assigned to placebo or citrulline malate (8 g; 60 minutes before the workout) groups and then performed repeated bouts of multiple lower body resistance exercise:

"Subjects warmed up on an upright stationary bike (Life Fitness, Brunswick Corporation, Lake Fores, IL) for five minutes, at 60 – 70 revolution/minute with a mass of 3 – 5 kg. Following this warm up, subjects performed two warm up sets (10 repetitions at 90.9 kg and 8 repetitions at 136.4 kg) on the leg press machine. Subjects rested three minutes between sets during the warm up and trial sets.

Next, 60% of each subject’s predetermined 1RM was loaded on the leg press machine and the subject completed as many repetitions as possible until failure occurred. This process was completed for 4 additional sets for a total of 5 sets on the leg press. Next, the subjects performed one warm upset (10 repetitions) on the hack squat machine at a mass of 40.9 kg. This warm up set was followed by 5 sets of 60% of their predetermined 1RM to failure. Finally, following one warm up set (10 repetitions at 36.4 kg) on the leg extension, subjects completed 5 sets of 60% of their 1RM to failure." (Wax. 2014)

The rest periods (recovery periods between sets of exercise), exercise order, and number of sets performed were the same for all subjects in this investigation, for sessions 2 and 3. Blood lactate, heart rate, systolic blood pressure, and diastolic blood pressure were determined pre and post exercise.
The exercise protocol resulted in sequential significant (p < 0.05) decrease in the number of repetitions in all three exercises. However, subjects in the citrulline malate group performed significantly (p < 0.05) higher number of repetitions during all three exercises compared to placebo group.

Figure 1: Wax et al. observed significant increases in maximal leg press, hack squat, and leg extension (not shown) repetitions in response to the ingestion of 8g of citrulline malate 60min before exercise (Wax. 2014)

Blood lactate and heart rate, on the other hand, were significantly increased (p < 0.05) post-exercise in both groups with no significant inter-group differences between citrulline malate and placebo (p > 0.05).
References:

• Briand, Joël, et al. "Use of a microbial model for the determination of drug effects on cell metabolism and energetics: Study of citrulline?malate." Biopharmaceutics & drug disposition 13.1 (1992): 1-22.
• Hickner, Robert C., et al. "L-citrulline reduces time to exhaustion and insulin response to a graded exercise test." Medicine and science in sports and exercise 38.4 (2006): 660-666.
• Giannesini, Benoît, et al. "Citrulline malate supplementation increases muscle efficiency in rat skeletal muscle." European journal of pharmacology 667.1 (2011): 100-104.
• Pérez-Guisado, Joaquín, and Philip M. Jakeman. "Citrulline malate enhances athletic anaerobic performance and relieves muscle soreness." The Journal of Strength & Conditioning Research 24.5 (2010): 1215-1222.
• Verleye, M., et al. "Effects of citrulline malate on bacterial lipopolysaccharide induced endotoxemia in rats." Arzneimittelforschung 45.6 (1995): E712.
• Wax, Benjamin, et al. "Effects of Supplemental Citrulline Malate Ingestion During Repeated Bouts of Lower-body Exercise in Advanced Weight Lifters." The Journal of Strength & Conditioning Research (2014).

A personal trainer who knows what hes doing is that he will push you exactly so far as it is necessary to make maximal progress. Interestingly, even the best trainers will fail doing the same for themselves.

The more is better mentality thats so characteristic of our lives in the Western world of affluence is imho the most important obstacle trainees all around the world meet on their way to increased muscle strength, size and performance. Against that background its a pity that the results of a recent study from the School of Kinesiology and Health Studies at the Queen’s University relate to sprint training, only. So, after having a look at Jason G. E. Zelt, I will briefly take a look at similar evidence from the more popular field of "working out to look good naked" and to be as strong as Superman.

But lets not waste any more time and sprint straight to the point! Zelt et al. published the results of the initially mentioned study in the peer-reviewed European Journal of Applied Physiology (Zelt. 2014).
As Zelt et al. point out, the purpose of this study was twofold: (1) to confirm that reductions in SIT work-interval duration do not result in reduced adaptations in aerobic and anaerobic capacity, and (2) to examine the effects of reduced work interval duration on submaximal determinants of exercise performance, namely lactate threshold and critical power.

In accordance with previous studies where aerobic capacity and aerobic performance were measured (Burgomaster. 2008; Hazell. 2010), the scientists hypothesized that reducing SIT work interval duration would have no effect on training-induced increases in lactate threshold and critical power.

Figure 1: Effects of high (SIT30) and lower volume (SIT15) training on power output and lactate threshold (Zelt. 2014)

And indeed, it hadnt. While there was a significant main effectof training on VO2peak such that VO2peak was elevated post-training, no significant difference was observed in the improvements observed between groups (ET ~13 %, SIT 30–4 %, SIT 15–8 %; not shown in Figure 1).

"A significant main effect of training was observed such that lactate threshold and critical power were higher during post-testing across all groups (p  <  0.05). There was a main effect of training (p  <  0.05) on Wingate peak power with no differences observed between groups at post training."

As the researchers point out, these results clearly indicate that "reducing SIT work-interval duration from 30 to 15 s had no impact on training-induced increases in aerobic or anaerobic power, or on increases in lactate threshold (absolute) and critical power."
The initially hinted at question that remains is yet: "Is this true for the more popular training goal of getting strong, ripped and buffed, as well?" The answer to this question is certainly not easy to answer, as it may easily depend on your training status, your exact goals and maybe even the body parts youre training. You dont get it? Well, I guess its best Ill provide you with a few examples:

• Look at his legs, Ronnie Coleman must have done something right... and guess what, the study at hand suggests that part of it could have been his insane training volume.

  the legs of advanced trainees may need a little more hammering -- Maybe you remember this being the take-home message from a previous article, i.e. "Advanced Trainees Benefit from Increased Training Volume! Greater & Steadier Strength Gains with 8 Sets of Squats. Plus: Over 6 Weeks, 1 Set and 4 Sets Equally (In-)Effective." | read more
• the classic single vs. three set debate is still not settled -- While Starky et al.s 1996 study is one of the studies that appears to tip the scale in favor of studies suggests that there is no significant different in the strength and muscle gains in response to increasing the number of sets on a given exercise from one to three sets. Unfortunately, Starky et al. as well as most of their successors picked untrained noobs to test their hypothesis. And we all know: Noobs grow from simply looking at a barbell, right?
  
  So what do other studies say? Studies that used subjects like you and me? People who have been training regularly for ten or more years? People like the fifty-one experienced (>3 years), trained junior lifters who were randomly assigned to low, medium and high volume resistance training in a 2005 study by Juan J. Gonzalez-Badillo et al.
  

  Figure 2: Number of repetitions per week and average intensity (AI) during the 10-week training period in the low-volume (LVG), moderate-volume (MVG), and high-volume (HVG) training groups (left); EEffect size for the snatch, clean & jerk, and squat performances. LVG low volume group; MVG medium volume group; HVG high volume group (Gonzalez-Badillo. 2005)

  As you can see in Figure 2 (right), there is a highly significant advantage of the medium vs. both the low and high volume group who trained at significantly lower, respectively higher volumes (see Figure 2, left) than the guys in the medium volume group for squats and clean & jerks. The snatch, on the other hand, probably because it is the most demanding exercise, technically speaking, benefited from a "little" more volume (~ 100 reps per week).
• the lower the volume, the higher the frequency -- Furthermore, the overview in Table 1, which was originally published as part of a review of the determinants of strength training success by Tan (1999) shows that another volume-related parameter, i.e. the training frequency, figures, as well; with high(er) frequencies producing greater increases in strength gains.

  Table 1: Summary of studies looking into optimal training frequency (Tan. 1999)

  Up to five training sessions for the upper and up to four for the lower body are possible, but whether theyre in fact as productive as the studies Tan cites would suggest appears questionable and will certainly depend on the volume of the individual sessions.
  
  The fact that it seems as if the upper body would respond more favorably to increases in training frequency than the lower body would albeit stand in line with the previously cited beneficial effects of high(er) volume training on the legs.
  
  

  Figure 3: Generally speaking a 2007 review of the literature by Mathias Wernbom et al. supports the notion that legs (in this case the quadriceps) dont just gain the most strength, but also the most size with ~3 training sessions per week; with the one outlier (12x/week) being a low intensity Kaatsu study by Abe et al. (2005)

  Why? Well, the study by Robins et al. conducted in 2012 (learn more) used a high volume on training days, but a necessarily low training frequency (two session per week, A + B). As Tan points out, ...
  

  "[...a]nother point to note from Table 1 is that previously trained athletes are closer to their strength potential and may require higher frequencies compared with untrained athletes" (Tan. 1999)."

  A statement that takes us back to the simple, but significant assessment that we cannot expect to find a training volume thats perfect for everyone: Individualization is key!

As far as the design of specific routines is concerned the previously cited review by Wernborn et al. offers a neat overview of suggested training principles, most of you will be familiar with.

Table 2: Recommendations for dynamic external resistance training for hypertrophy (Wernborn. 2007)

If you take a look at the middle column, you will hopefully realize that the suggested progression from 1-2 sets to 3-6 sets is specified on a "per muscle group" basis. The three biceps exercise with three sets of 8-12 reps, each, are thus off the charts, already; and youd be better off doing three sets of barbell curls and nosebreakers to end up at your maximum of six sets per body part (in this case the "arms") to return to the gym two days later and hammer your arms with three sets of hammer curls and cable press-downs.
References:

• Abe, Takashi, Charles F. Kearns, and Yoshiaki Sato. "Muscle size and strength are increased following walk training with restricted venous blood flow from the leg muscle, Kaatsu-walk training." Journal of Applied Physiology 100.5 (2006): 1460-1466.
• Robbins, Daniel W., Paul WM Marshall, and Megan McEwen. "The effect of training volume on lower-body strength." The Journal of Strength & Conditioning Research 26.1 (2012): 34-39.
• Tan, Benedict. "Manipulating resistance training program variables to optimize maximum strength in men: a review." The Journal of Strength & Conditioning Research 13.3 (1999): 289-304.
• Wernbom, Mathias, Jesper Augustsson, and Roland Thomeé. "The influence of frequency, intensity, volume and mode of strength training on whole muscle cross-sectional area in humans." Sports Medicine 37.3 (2007): 225-264.
• Zelt, Jason GE, et al. "Reducing the volume of sprint interval training does not diminish maximal and submaximal performance gains in healthy men." European journal of applied physiology (2014): 1-10.

Yogurt & Co are good for athletes. But is this due to the bacteria?

As a regular here at the SuppVersity, you are well aware of the far-reaching effects of having the "right" or "wrong" bacteria in your tummy. The claim that having the "right" bacterial make-up in the gut could have beneficial effects on your performance in the gym or on the track is something you should not believe too credulously. If we had a study to prove these benefits, things would be different, though - a study like the one a group of researchers from the National Taiwan Sport University are about to publish in the scientific bible of resistance training, the Journal of Strength and Conditioning Research (Hsu. 2014).

In said study which is still available only as an accepted manuscript, Hsu et al. investigated the association of intestinal bacteria and exercise performance in specific pathogen-free (SPF), germ-free (GF), and Bacteroides fragilis (BF) gnotobiotic mice (animals in which only certain known strains of bacteria and other microorganisms are present).
To this ends, the scientist had the rodents swim to exhaustion to determine whether enteric bacteria alter antioxidant enzyme levels, exercise performance and physical fatigue. In addition, they tested the antioxidant enzyme activities, physical performance and anti-fatigue function after monocolonizing GF mice with B. fragilis (BF).

Figure 1: Time to exhaustion during exercise test and body composition of the miceaccording to the make-up of their gut microbiome; data expressed relative to group means (Hsu. 2014)

What they found was an increased time to exhaustion for the previously germ-free (GF) mice after theyd been colonized with B. fragilis (BF). Similar results were observed for the specific pathogen-free and Bacteroides fragilis gnotobiotic mice, who were also more enduring than the obesity resistant (Bäckhed. 2007) germ-free mice.
As any SuppVersity veteran would expect, the germ-free mice were leaner than the rest of the pack. At the same time, the "sterile" mice did yet also have a lower liver, muscle, brown adipose, and epididymal fat pad weight than the SPF and BF mice.

Figure 2: Selected markers of antioxidant status in serum and liver; expr. rel. to group means (Hsu. 2014)

The markers of antioxidant defenses, the scientists measured were highest in the lean + light germ-free mice and their specific pathogen-free cousins. Against that background its at least somewhat surprising that the performance differences are pretty significant.
References:

• Bäckhed, Fredrik, et al. "Mechanisms underlying the resistance to diet-induced obesity in germ-free mice." Proceedings of the National Academy of Sciences 104.3 (2007): 979-984.
• Cani, Patrice D., et al. "Gut microbiota fermentation of prebiotics increases satietogenic and incretin gut peptide production with consequences for appetite sensation and glucose response after a meal." The American journal of clinical nutrition 90.5 (2009): 1236-1243.
• Cox, Amanda J., et al. "Oral administration of the probiotic Lactobacillus fermentum VRI-003 and mucosal immunity in endurance athletes." British Journal of Sports Medicine 44.4 (2010): 222-226. 
• DiMarco, N. M., et al. "A–Z of nutritional supplements: dietary supplements, sports nutrition foods and ergogenic aids for health and performance—Part 30." British journal of sports medicine 46.4 (2012): 299-300.
• Gleeson, Michael, et al. "Daily probiotics (Lactobacillus casei Shirota) reduction of infection incidence in athletes." (2011). 
• Hsu et al. "Effect of Intestinal Microbiota on Exercise Performance in Mice." Journal of Strength and Conditioning Research.  DOI: 10.1519/JSC.0000000000000644 | Publish Ahead of Print.
• Martarelli, Daniele, et al. "Effect of a probiotic intake on oxidant and antioxidant parameters in plasma of athletes during intense exercise training." Current microbiology 62.6 (2011): 1689-1696.
• West, Nicholas P., et al. "Lactobacillus fermentum (PCC®) supplementation and gastrointestinal and respiratory-tract illness symptoms: a randomised control trial in athletes." Nutrition journal 10.1 (2011): 30.

The study at hand used plain ascorbic acid, no quack supplements with "advanced vitamin C".

While people tend to believe that vitamin C is good for anything, the evidence that it actually does anything good is relatively scarce. Against that background I am happy to tell you that a group of Greek researchers from the School of Physical Education and Sport Science, the European University Cyprus and theAristotle University of Thessaloniki have now finally confirmed what many of you probably thought was a long-established fact: "[L]ow vitamin C concentration is linked with decreased physical performance and increased oxidative stress and that vitamin C supplementation decreases oxidative stress and might increase exercise performance only in those with low initial concentration of vitamin C." (Paschalis. 2014)
When they came up with the study design, Paschalis et al. simply assumed that the mythical ergogenic effect of vitamin C actually existed. To test this hypothesis, they screened 100 males for vitamin C baseline values in blood, picked the 10 individuals with the lowest and the 10 with the highest vitamin C values from their baseline sample and assigned them to two groups.

Figure 1: Overview of the study design (Paschalis. 2014)

Using a placebo-controlled crossover design, the 20 selected subjects performed aerobic exercise to exhaustion (oxidant stimulus) before and after vitamin C supplementation for 30 days.

An overview of the study design is shown in Fig. 1. All measurements were performed between 08:00 and 11:00 h after overnight fasting. Initially, to examine whether rest ing blood vitamin C concentration affects aerobic perfor mance, VO2max was assessed (using incremental cycling test to volitional exhaustion) and was compared in both the low and the high vitamin C groups (Monark, Vansbro, Swe den). More specifially, the protocol started with a 50 W load at 50 rpm and increased by 10 W every 2 min until volitional fatigue. The test was terminated when three of the following four criteria VO2max were met: (1) volitional fatigue, (2) a lower than 2 mL/kg/min increase in VO2 despite an increase in workload, (3) a respiratory exchange ratio greater than or equal to 1.10, and (4) heart rate within 10 bpm of the predicted maximal heart rate (220–age). Res piratory gas variables were measured using a metabolic cart (Quark b2, Cosmed, Italy), which was calibrated before each test using standard gases of known concentration. The VO 2max assessment was used as a reference value to cal culate the workload at the relative intensity of each subject and ensured that all subjects would cycle at similar relative intensity during the following aerobic exercise sessions.

After the baseline testing had been done, the subjects within both the low and the high vitamin C groups received either placebo (3x333mg of lactose) or vitamin C supplementation (3x333mg of vitamin C), in a double-blind randomized crossover fashion (see Figure 1).

Figure 2: Changes in VO2max (left) and redox status (right) in subjects according to initial vitamin C status before and after vitamin C supplementation for 30 days (Paschalis. 2014).

As you can see in Figure 2 there were measurable differences in the response to the acute exhaustive exercise protocol (an oxidant stimulus), the subjects in both groups performed before and after vitamin C or placebo supplementation for 30 days. The data in Figure 2 does yet also show that the subjects who had been randomly assigned to the vitamin C supplement group had lower baseline VO2max levels. A fact that raises the question whether this is the result of a lower vitamin C intake or whether the vitamin C intake correlates with an unhealthier lifestyle that left the subjects unfit and with low vitamin C levels.
Unfortunately, this question is hard to answer based on the available research on vitamin C. While we have conflicting results with respect to its ability to impair the adaptational response to exercise (Close. 2014), there is very little evidence that it will actually have beneficial effects on any meaningful performance parameters. In fact, a study by Huck et al. that was published in the scientific journal Nutrition in 2013 is probably what comes closest to the results of the study at hand.

Figure 3: Effects of 500mg vitamin C per day on selected parameters in a 4 week chronic exercise + diet supplementation in obese men and women (Huck. 2013)

In said study Huck et al. observed that the provision of 500mg of vitamin C as an adjunct to exercise and diet in obese individuals lead to significant reductions in heart rate and the ratings of perceived exertion during exercise. The data in in Figure 3 does yet also tell you that there were no beneficial effects on VO2max, which best reflects the adaptational response to exercise.

This results of stands in contrast to the study at hand, but in line with previous results of studies in athletes, where only more or less irrelevant reductions of the acute inflammatory response to exercise were observed (Nieman. 2000; Peters. 2001; Tauler. 2002). A response of which you as a SuppVersity reader know that it is an essential part of the signalling cascade that triggers the adaptational response to. If we eventually get back to the Paschalis study, it would thus appear that athletes who are usually consuming more than enough vitamin C in their diets and are not at particular risk of developing low serum vitamin C levels would see similar results as the "high vitamin C" subjects in the Paschalis study, i.e. none - even worse, in view of the potential negative effects on the training induced adaptations that could not become visible in the study at hand, because there was no exercise protocol involved, it could even harm their progress.
References:

• Close, G. L., and M. J. Jackson. "Antioxidants and exercise: a tale of the complexities of relating signalling processes to physiological function?." The Journal of physiology 592.8 (2014): 1721-1722.
• Huck, Corey J., et al. "Vitamin C status and perception of effort during exercise in obese adults adhering to a calorie-reduced diet." Nutrition 29.1 (2013): 42-45.
• Nieman, David C., et al. "Influence of vitamin C supplementation on cytokine changes following an ultramarathon." Journal of Interferon & Cytokine Research 20.11 (2000): 1029-1035.
• Paschilis, V. et al. "Low vitamin C values are linked with decreased physical performance and increased oxidative stress: reversal by vitamin C supplementation." Eur J Nutr (2014). Ahead of print.
• Paulsen, Gøran, et al. "Vitamin C and E supplementation hampers cellular adaptation to endurance training in humans: a double?blind, randomised, controlled trial." The Journal of physiology 592.8 (2014): 1887-1901.
• Peters, E. M., et al. "Vitamin C supplementation attenuates the increases in circulating cortisol, adrenaline and anti-inflammatory polypeptides following ultramarathon running." International journal of sports medicine 22.7 (2001): 537-543.
• Picklo, Matthew. "Supplementation with vitamin E and vitamin C inversely alters mitochondrial copy number and mitochondrial protein in obese, exercising rats (1030.5)." The FASEB Journal 28.1 Supplement (2014): 1030-5. 
• Powers, Scott K., et al. "Dietary antioxidants and exercise." Journal of sports sciences 22.1 (2004): 81-94.
• Powers, Scott K., And Kurt J. Sollanek. "Endurance Exercise And Antioxidant Supplementation: Sense Or Nonsense?-Part." Sports Science 27.137 (2014): 1-4.
• Tauler, P., et al. "Diet supplementation with vitamin E, vitamin C and ?-carotene cocktail enhances basal neutrophil antioxidant enzymes in athletes." Pflügers Archiv 443.5-6 (2002): 791-797.